2019-nCoV lyrics

Superoxide (O2) and hydrogen peroxide (H2O2) are more selective in their reactions with biological molecules
While singlet oxygen is more potent, it might not be as selective. Could be more dangerous
Oxygen binds to hemoglobin at the ferrous iron. The ferrous state (Fe2+) of iron is a condition for hemoglobin normal function
Mathemoglobin reduces Ferric Form Iron back to Fe2+ and goes back to being a hemoglobin
Thus hemoglobin auto-oxidation causes superoxide formation within erytrocyte.
­ O2 reacts with itself generating molecular oxygen (O2) and hydrogen peroxide (H2O2) a source for hydroxyl radical.­ O2 reacts fast with NO• radical generating toxic peroxinitrite (ONOO−), a reactive nitrogen specie (RNS). At physiological pH, ONOO− rapidly protonates to peroxynitrous acid, ONOOH. This powerful oxidizing and nitrating agent can directly damage proteins and lipids.
Thus, any system producing O2 and NO• can cause biological damage, and erythrocytes make no exception.
The hydroxyl radical (OH•), which is highly reactive, diffuses only a short distance before it reacts with whatever biomolecules it collides with.
Being so harmful cells carefully control hydroxyl radical by limiting the availability of both Fe2+ and H2O2.

Erythrocytes are particularly affected by oxidation of polyunsaturated fatty acids in membrane phospholipids which causes their peroxidation, degradation and fragmentation . During lipid peroxidation other reactive species as peroxyl radicals are generated in a succession of chain reactions. This reactive intermediates amplify the injury at the place were they are formed. In nucleus containing endothelium cell beside lipids and proteins oxidation ROS cause DNA injuries. Free radicals can interact with DNA leading to strand breaks or structural changes such as adduct formation. So oxydative attacks are neither good nor bad as they may help the cell survive or trigger its death.

NO2 nitrogen dioxide or nitrite. In organism is found in its corresponding salts nitrites( from nitrous acid HNO2)
NO3 nitrate (from nitric acid HNO3) also found in the body in corresponding salts
Nitrogen derivatives convert into each other forward and backward continuously under shifting conditions within cells so KNO3 administered in safe non-lethal dosage might produce more available nitrates for the reaction since it's the safest and most edible of the nitrates.

Generated NO is a gaseous molecule with unpaired electrons and as a consequence a radical; it is lipophilic and diffuses rapidly through membranes. NO is a messenger in many physiological processes: endothelial relaxation of the smooth muscle, inhibition of platelet aggregation, neurotransmission and cytoxicity. NO pathology includes both low and high concentrations as follows: insufficient NO production is involved in hypertension, the activation of platelet aggregation and atherogenesis while high NO production generates septic shock, stroke, and carcinogenesis. Therefore the doses administered have to be below the threshold and systematic. To summarize NO can be produced by eNOS from either L-arginine in good oxygenation physiological state or from nitrite in hypoxia. In vitamins deficiency (low BH4 levels) eNOS produces superoxide radical. Recent studies demonstrate that endothelium cells surprisingly produce ROS under hypoxia
Capsule form of a mixture of the potassium nitrate salt with vitamins B12 and B6 complex along with a multivitamin administered daily would in theory produce a safe auto-oxydation which may keep a healthier red blood cell and help it get rid of infectors through the process of auto-oxydation systemically, these responses enhance the delivery of O2 to cells and facilitate the production of glycolytic ATP instead of mitochondrion, also included in the pill acetaminophen to combat the fever and promethazine to combat the nausea symptoms. The virus will ultimately die for being unable to protrude the red blood cells leading to the present amount of white blood cell to be sufficient to combat the virus.
Leukopenia and lymphopenia seem to be the common trend in 2019-nCoV and those are deficiencies common of those with low white blood cells or unhealthy white blood cells.
The recommended drug treatment according to some popular sites were cinanserin and cyclosporine. However those two drugs are white blood cell suppressants and neurotransmitter blockers. What is more likely to happen is that the white blood cells would fail to defend if suppressed and that the neurotransmitter blockers will cause even more adverse health effects such as immobility and neuropathy.


(Sources: https://www.intechopen.com/books/blood-cell-an-overview-of-studies-in-hematology/homocysteine-in-red-blood-cells-metabolism-pharmacological-approaches)